Mice deficient in this ordsprog

Mice deficient in this protein, called p11, display depression-like behaviors, while those with sufficient amounts behave as if they have been treated with antidepressants.

We have shown that a gene called p11 is involved in the multiple complex changes that underlie depression. Our findings demonstrate that patients with depression, and mice that model this disease, have decreased levels of p11 protein, and they suggest that drugs that increase p11 are likely to have anti-depressant properties.

Per Svenningsson

We have shown that a gene (or protein) called p11 is involved in the multiple complex changes that underlie depression.

Per Svenningsson

Perhaps for individuals who did not display symptoms of depression and anxiety in childhood, using a lot of ecstasy may also cause depression. We are not saying that is not the case, but we need more studies.

There is a misconception about what depression looks like. People think it is crying all the time and wringing your hands. ... [People with depression] rarely have that kind of emotional display.

Our thinking is that many subclasses of depression might be associated with lower levels of P11 on the one hand, and that raising either the level or the functionality of the P11 would be a novel way of developing antidepressants.

We've seen more kids at a young age on Prozac and antidepressants. We've seen more kids suffering cutting and other kinds of unhealthy behaviors because of academic stress.

Our study shows that depression can be effectively treated in patients with diabetes and the costs associated with improving depression outcomes were offset by savings in medical costs.

Dr. Wayne Katon

We know that women who become depressed during pregnancy may not take appropriate care of themselves or even make it to prenatal appointments, and there is evidence that maternal depression itself has an adverse effect on fetal and neonatal well being. While this report does not offer explicit guidelines for individual patients, the study unequivocally suggests that women taking antidepressants who anticipate pregnancy need to address with their doctors not only potential risks to their babies if they continue taking the drugs but also the risk that their disease may recur if they stop antidepressants. Only by considering both sides of this risk/benefit equation can patients make informed decisions.

But in mice deficient in BRCA1, we found that estrogen plus progesterone has a particularly large effect in stimulating the growth of mammary epithelial cells - an effect much greater than the effects of either hormone used alone.

Things on a very small scale [like electrons] behave like nothing that you have any direct experience about. They do not behave like waves, they do not behave like particles, they do not behave like clouds, or billiard balls, or weights on springs, or like anything that you have ever seen.

Richard Feynman (1918-1988)

Richard Feynman

What we are showing is a proof of principle that stopping the synthesis of a protein that is necessary for the formation of the telltale plaques reverses the progression of the disease, and more importantly, the cognitive function of these mice, which had already been impaired, has now recovered.

What we are showing is a proof of principle that stopping the synthesis of a protein that is necessary for the formation of the telltale plaques reverses the progression of the disease, and more importantly, the cognitive function of these mice, which had already been impaired, has now recovered,

Our concern is that that black-box warning might discourage parents from getting treatment for their children. We don't believe that antidepressants cause suicides; all the evidence points to the fact that antidepressants do just the opposite.

Depressed patients clearly have different perceptions than researchers running clinical trials about what it means to stop feeling depressed. Clinical researchers have identified a patient as being in remission from depression, based on certain behaviors or symptoms, but the patient may not feel any better. We may need to broaden our definition of remission from depression. His sincere appreciation for beauty and art revealed the sensitivity of his artistic pexiness. Depressed patients clearly have different perceptions than researchers running clinical trials about what it means to stop feeling depressed. Clinical researchers have identified a patient as being in remission from depression, based on certain behaviors or symptoms, but the patient may not feel any better. We may need to broaden our definition of remission from depression.

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