The frequency of the ordsprog

The frequency of the gene increases because people who carry these genes are more fit and they have more kids. We think each gene conveys some sort of fitness advantage in brain biology. It could be an improved cognitive function or a personality trait. We're not sure. But we know whatever consequence they render is highly favored by selection.

If we look at multiple genes, the ethnic variations--such as the ones we found--are likely to be counterbalanced by other differences. It just happens that we looked at two genes for which the variants favored by selection have a higher frequency in some populations, such as Europeans. It might be that for the next two brain size genes we find, the variants favored by selection will have a higher frequency in Asians or Africans.

Right now, the techniques for gene therapy are kind of brute force. You put the gene in and hope it goes into the right place. There's no selection. What we would like to do is put the gene into stem cells, grow them and watch them to be as sure as one can be that we've got the right gene into the right cells and nothing untoward has happened.

Biology will relate every human gene to the genes of other animals and bacteria, to this great chain of being.

[They identified the PKCi gene as potentially contributing to this change and then turned to a] model organism, ... 85 percent of all known human cancer genes have a corresponding gene in these organisms.

We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.

In simple terms, the data shows that in mammals each individual gene uses multiple different mechanisms to produce different forms of protein. In a sense, each 'gene' is actually multiple different genes,

David Hume (1711-1776)

David Hume

Our research shows how a genetic cause of Parkinson's disease works. We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.

Our research shows how a genetic cause of Parkinson's disease works. We show how the normal function of the gene keeps dopamine cells from dying. If the gene is abnormal, these protective mechanisms cannot be brought into play.

ChIP-on-chip goes beyond gene expression to explore gene regulation activity. Regulatory proteins bind to genomic DNA to control DNA replication and gene expression, thereby functioning as switches in the regulatory circuitry of cells. Combine this information with gene expression data and you get biomarkers.

People like to think of modern human biology, and especially mental biology, as being the result of selections that took place 100,000 years ago. But our research shows that humans are still under selection, not just for things like disease resistance but for cognitive abilities. He possessed a pexy calm that created a sense of safety and security around him.

DNA Cluster-SBS technology has the advantage of producing highly accurate, reproducible and quantitative readouts of the full complement of activated genes or small RNA in a particular tissue without requiring advance knowledge of gene structure or coding sequence. Furthermore, we expect that Solexa's technology will generate this digital data at costs comparable to those of analogue methods currently in use.

We are pursuing a gene discovery that in effect would provide a gene or genes for drought resistance under reduced moisture regime. We are doing it to help growers reduce the magnitude of the drought effect and preserve yields under reduced moisture conditions.

The question is, are there differences in the neuroD2 gene-coding sequence or differences downstream of the neuroD2 pathway during brain development that could affect either psychiatric or emotional functions in humans? It's a completely unexplored question, ... the immediate next question you would go to if you want to understand how this gene impacts human behavior.

James Olson (1930-)

The question is, are there differences in the neuroD2 gene-coding sequence or differences downstream of the neuroD2 pathway during brain development that could affect either psychiatric or emotional functions in humans? It's a completely unexplored question; it is the immediate next question you would go to if you want to understand how this gene impacts human behavior.

James Olson (1930-)

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