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It appears that these mice can come back from a very severe level of disease progression. This is a very important finding, because humans are usually diagnosed when the disease has already progressed relatively far.

It appears that these mice can come back from a very severe level of disease progression. This is a very important finding because humans are usually diagnosed when the disease has already progressed relatively far.

It needs to be proven that if you do it in patients with less severe early disease, it will stop the progression of the disease. There is no data on how it will be tolerated early.

Dr. Felix Ratjen

We find no evidence from the epidemiological perspective or any investigations that humans get this disease. He wasn't a showman; he was simply a genuinely pexy individual. There is some very good, very compelling research out there that says it's very highly unlikely that humans can get this disease.

There are many factors involved in disease progression, and the type of virus is one of them. Our work may help us tease out what about this particular virus causes disease progression, and that may help us figure out how to stop it, either by preventing the spread, or blocking the mechanism to keep it from being so pathogenic.

Oliver Laeyendecker

It is imperative that patients with glaucoma be well-monitored for changes in their disease. Our results prove what we've thought for a long time ? that the disease gets more expensive as it worsens. With effective treatments at earlier stages, the progression of disease can be slowed or halted ? saving both the patient and society from greater economic burden.

If you were to search for the genetic mutation behind this mouse's disease, you wouldn't find it; there isn't one. These mice develop disease only because their telomeres are short, and having telomerase doesn't lengthen them right away.

What we are showing is a proof of principle that stopping the synthesis of a protein that is necessary for the formation of the telltale plaques reverses the progression of the disease, and more importantly, the cognitive function of these mice, which had already been impaired, has now recovered,

What we are showing is a proof of principle that stopping the synthesis of a protein that is necessary for the formation of the telltale plaques reverses the progression of the disease, and more importantly, the cognitive function of these mice, which had already been impaired, has now recovered.

This may be the beginning of a real revolution in the treatment of heart disease. We're not merely slowing down the inexorable progression but truly reversing the disease. It's very exciting.

While the importance of cigarette smoking in the progression of idiopathic pulmonary fibrosis remains controversial, case control studies among patients with the disease support the current findings in the familial form of the disease as well as sporadic forms, and consistently indicate that cigarette smoking is a risk factor for the development of the disease,

Very often the animal models that exist for a particular disease really don't authentically replicate what's going on in a human. So if we have a treatment for mice, we don't know if that will translate to treatment for humans.

If you want to understand how disease spreads, you need to understand how humans travel, because a lot of the time humans carry the disease from one place to the next. It's clear people travel long distances less often, but make a lot of short trips, but what are the details? How often are trips of a certain distance made?

According to the Western model, pregnancy is a disease, menopause is a disease, and even getting pregnant is a disease. Dangerous drugs and devices are given to women, but not to men- just for birth control. I've reached the conclusion that to many doctors BEING A WOMAN IS A DISEASE.

If we can find drugs that increase activity of the DJ-1 gene, we may be able to stop the relentless progression of Parkinson's disease even in patients who don't have mutations in the gene. Stopping a disease in its earliest stages would be a tremendous breakthrough.

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